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Carbone, Michele
One or more keywords matched the following items that are connected to Carbone, Michele
Item TypeName
Academic Article Activated cAMP response element binding protein is overexpressed in human mesotheliomas and inhibits apoptosis.
Academic Article Cancer cell secretion of the DAMP protein HMGB1 supports progression in malignant mesothelioma.
Academic Article High Incidence of Somatic BAP1 alterations in sporadic malignant mesothelioma.
Academic Article Preclinical development of HIvax: Human survivin highly immunogenic vaccines.
Academic Article HMGB1 targeting by ethyl pyruvate suppresses malignant phenotype of human mesothelioma.
Academic Article BAP1 regulates IP3R3-mediated Ca2+ flux to mitochondria suppressing cell transformation.
Academic Article Monoubiquitination of ASXLs controls the deubiquitinase activity of the tumor suppressor BAP1.
Academic Article SV40 infection induces telomerase activity in human mesothelial cells.
Academic Article Different susceptibility of human mesothelial cells to polyomavirus infection and malignant transformation.
Academic Article Cross reactivity between many anti-human antibodies for their hamster homologs provide the tools to study the signal transduction pathway activated by asbestos and SV40 in the malignant mesothelioma model.
Academic Article Crocidolite asbestos and SV40 are cocarcinogens in human mesothelial cells and in causing mesothelioma in hamsters.
Academic Article Identification of intelectin overexpression in malignant pleural mesothelioma by serial analysis of gene expression (SAGE).
Academic Article TNF-alpha inhibits asbestos-induced cytotoxicity via a NF-kappaB-dependent pathway, a possible mechanism for asbestos-induced oncogenesis.
Academic Article Activated TNF-alpha/NF-kappaB signaling via down-regulation of Fas-associated factor 1 in asbestos-induced mesotheliomas from Arf knockout mice.
Concept Cells, Cultured
Concept Tumor Cells, Cultured
Academic Article Asbestos induces mesothelial cell transformation via HMGB1-driven autophagy.
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